Alexander Disease – Page 2 – Waisman Center – UW–Madison

Regulation of GFAP Expression

Type II Alexander disease caused by splicing errors and aberrant overexpression of an uncharacterized GFAP isoform

GFAP at 50

Refining the concept of GFAP toxicity in Alexander disease.

AP-1 and the injury response of the GFAP gene.

Mutations in GFAP Disrupt the Distribution and Function of Organelles in Human Astrocytes.

Tissue and cellular rigidity and mechanosensitive signaling activation in Alexander disease

Antisense suppression of glial fibrillary acidic protein as a treatment for Alexander disease

Glial fibrillary acidic protein exhibits altered turnover kinetics in a mouse model of Alexander disease

Characterization of a panel of monoclonal antibodies recognizing specific epitopes on GFAP.